Test Bank Docx Ch.4 Stabilizing Long-Term Potentiation 3e - Neurobiology Learning 3e | Test Bank Rudy by Jerry W. Rudy. DOCX document preview.

Test Bank Docx Ch.4 Stabilizing Long-Term Potentiation 3e

Chapter 4: Stabilizing Long-Term Potentiation

Test Bank

Type: true-false

Title: Chapter 4 - Question 1

1. Application of a low-frequency stimulus within a day after the TBS-inducing stimulus is sufficient to interrupt the development of a long-lasting LTP.

a. True

b. False

Type: true-false

Title: Chapter 4 - Question 2

2. Low-frequency stimulation delivered 15 minutes after LTP induction has no effect on stabilization

a. True

b. False

Type: true-false

Title: Chapter 4 - Question 3

3. When cofilin is phosphorylated, F-actin is more likely to remain in its filament state.

a. True

b. False

Type: true-false

Title: Chapter 4 - Question 4

4. Applying drugs that inhibit myosin IIb prior to TBS prevents the induction and stabilization of LTP.

a. True

b. False

Type: true-false

Title: Chapter 4 - Question 5

5. When cofilin is phosphorylated actin polymerization is prevented.

a. True

b. False

Type: true-false

Title: Chapter 4 - Question 6

6. Neural cadherins can exist as either monomers or cis-stranded dimers, with equal adhesive properties.

a. True

b. False

Type: true-false

Title: Chapter 4 - Question 7

7. Neutralizing the response of β integrins to extracellular ligands prevents enduring LTP normally produced by TBS, but it has no effect on the initial induction of LTP.

a. True

b. False

Type: true-false

Title: Chapter 4 - Question 8

8. Large spines endure longer than small spines.

a. True

b. False

Type: true-false

Title: Chapter 4 - Question 9

9. When cofilin is phosphorylated actin polymerization is prevented.

a. True

b. False

Type: true-false

Title: Chapter 4 - Question 10

10. Drugs like latrunculin and cytochalasin prevent actin polymerization.

a. True

b. False

Type: true-false

Title: Chapter 4 - Question 11

11. In it nonactive state CaMKII bundles active filament.

a. True

b. False

Type: true-false

Title: Chapter 4 - Question 12

12. In its non-phosphorylated state cofilin can sever actin filaments.

a. True

b. False

Type: true-false

Title: Chapter 4 - Question 13

13. When actin polymerization is inhibited the actin cytoskeleton will enlarge.

a. True

b. False

Type: true-false

Title: Chapter 4 - Question 14

14. Inhibiting NMDA receptors will prevent spine enlargement.

a. True

b. False

Type: true-false

Title: Chapter 4 - Question 15

15. Stabilization of LTP depends on the regulation of actin dynamics.

a. True

b. False

Type: true-false

Title: Chapter 4 - Question 16

16. The generation but not the stabilization of LTP depends on the reorganization of N-cadherins.

a. True

b. False

Type: true-false

Title: Chapter 4 - Question 17

17. The stabilization of LTP depends on the reorganization of N-cadherins, but the generation of it does not.

a. True

b. False

Type: true-false

Title: Chapter 4 - Question 18

18. Applying latrunculin and cytochalasin to the LTP slice 15 minutes following induction has no effect on LTP stabilization.

a. True

b. False

Type: true-false

Title: Chapter 4 - Question 19

19. The delivery of low-frequency stimulation prior to induction of LTP has no effect on its generation but prevents LTP stabilization.

a. True

b. False

Type: essay/short answer question

Title: Chapter 4 - Question 20

20. Explain the observation that people with amnesia resulting from an accident often can recall events that took place long ago but not events that occurred within minutes of the accident?

Type: essay/short answer question

Title: Chapter 4 - Question 21

21. How can a weak stimulus (low frequency) be used to prevent the development of a long-lasting LTP?

Type: essay/short answer question

Title: Chapter 4 - Question 22

22. What is a benefit of larger spines?

Type: essay/short answer question

Title: Chapter 4 - Question 23

23. Identify a role for inactive CaMKII.

Type: essay/short answer question

Title: Chapter 4 - Question 24

24. Why is non-phosphorylated cofilin important to the subsequent enlargement of the spine?

Type: essay/short answer question

Title: Chapter 4 - Question 25

25. What is the role of LIMK in the stabilization of LTP?

Type: essay/short answer question

Title: Chapter 4 - Question 26

26. In Harou Kasai’s study of single spine enlargement, what was the effect of antagonizing NMDA receptors?

Type: essay/short answer question

Title: Chapter 4 - Question 27

27. In Harou Kasai’s study of single spine enlargement, what was the effect of the actin polymerization inhibitor, latrunculin

Type: essay/short answer question

Title: Chapter 4 - Question 28

28. The severing properties of cofilin are initially important to stabilization. However, it is important that this function be terminated. How does this normally happen?

Type: essay/short answer question

Title: Chapter 4 - Question 29

29. The severing properties of cofilin are initially important to stabilization. However, it is important this this function be terminated. Why?

Type: essay/short answer question

Title: Chapter 4 - Question 30

30. Describe how actin filaments are broken down into smaller units. Why is this beneficial?

Type: essay/short answer question

Title: Chapter 4 - Question 31

31. What is an “adhesive zipper”?

Type: essay/short answer question

Title: Chapter 4 - Question 32

32. Briefly describe how the delivery of low-frequency stimulation prior to induction of LTP affects its generation and its stabilization.

Document Information

Document Type:
DOCX
Chapter Number:
4
Created Date:
Aug 21, 2025
Chapter Name:
Chapter 4 Stabilizing Long-Term Potentiation
Author:
Jerry W. Rudy

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