Quantitative and Qualitative Platelet | Full Test Bank Ch.16 - Test Bank | Hematology in Practice 3e by Betty Ciesla by Betty Ciesla. DOCX document preview.
Chapter 16. Quantitative and Qualitative Platelet Disorders
Multiple Choice
1. A decreased platelet count is most often anticipated when a patient demonstrates:
A. Rectal bleeding
B. Epistaxis
C. Genitourinary bleeding
D. Severe hemorrhage
L.O. 16.2
2. Increased platelets are seen in which of the following disorders?
A. ITP
B. TTP
C. ET
D. vWD
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3. Which of the following causes irreversible inactivation of platelets?
A. Heparin
B. Coumadin
C. Aspirin
D. Vitamin K
L.O. 16.3
4. A 2-year-old boy develops the following symptoms: petechiae, purpura, and nosebleeds. The platelet count is 18,000. The most likely explanation is:
A. Drug-induced thrombocytopenia
B. Secondary thrombocytopenia
C. Idiopathic thrombocytopenic purpura
D. Thrombotic thrombocytopenic purpura
L.O. 16.8
5. Which the following test results is normal in a patient with classic von Willebrand’s disease?
A. Bleeding time
B. Activated partial thromboplastin time
C. Platelet count
D. Factor VIII and vWF levels
L.O. 16.5
6. The autoantibody generated in ITP is directed against:
A. vWF
B. Collagen
C. GP IIb/IIIa, GP Ib/IX
D. Fibrinopeptides A and B
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7. Which of the following are platelet adhesion disorders?
A. von Willebrand’s disease and Glanzmann’s thrombasthenia
B. von Willebrand’s disease and Bernard-Soulier syndrome
C. Glanzmann’s thrombasthenia and Bernard-Soulier syndrome
D. Glanzmann’s thrombasthenia and congenital fibrinogenemia
L.O. 16.5
8. Immune drug-induced thrombocytopenia is a result of all of the following drugs except:
A. NSAIDs
B. Heparin
C. Amoxicillin
D. Aspirin
L.O. 16.6
9. In thrombotic thrombocytopenic purpura, which of the following statements is the most accurate?
A. It is associated with E. coli O157:H7.
B. It is associated with fever, renal failure, and neurological complications.
C. The organ most affected is the kidney.
D. More males are affected than females
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10. The presence of thrombocytopenia and giant platelets on the peripheral smear of a patient is characteristic of:
A. Classic von Willebrand’s disease
B. Hemophilia A
C. Glanzmann’s thrombasthenia
D. Bernard-Soulier syndrome
L.O. 16.5
11. Decreased platelets in combination with bloody diarrhea, vomiting, and abdominal pain is seen in which of the following?
A. HUS
B. ITP
C. Glanzmann’s thrombasthenia
D. TTP
L.O. 16.9
12. In hemolytic uremic syndrome (HUS), which of the following statements is the most accurate?
A. The organ most affected is the spleen.
B. The toxin responsible for this syndrome is produced by E. coli O157:H7.
C. Impaired neurological function is one of the primary symptoms.
D. Platelets are permanently impaired.
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13. Platelet aggregation cannot occur if _______ is absent.
A. vWF
B. GP IIb/IIIa
C. antibodies
D. GP Ib/IX
L.O. 16.5
14. All of the following are associated with platelet disorders except:
A. Thrombocytopenia
B. Deep muscle hemorrhages
C. Epistaxis and menorrhagia
D. Abnormal platelet morphology
L.O. 16.1
15. All of the following can affect the platelet count except:
A. Improper mixing of specimen
B. Not using a tourniquet
C. Presence of EDTA-antibodies
D. Not using anticoagulated tubes
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NARRBEGIN:
NARREND
16. Red cell morphology such as the one at the tip of the pointer are associated with:
A. von Willebrand’s disease
B. Glanzmann’s thrombasthenia
C. Thrombotic thrombocytopenic purpura
D. All of the above
NAR: Figure 16-1
L.O. 16.10
17. Thrombocytosis is defined as a platelet count that is:
A. Greater than 450 × 109/L
B. Less than 450 × w109/L
C. Greater than 600 × 109/L
D. Less than 300 × 109/L
L.O. 16.1
18. Aspirin therapy irreversibly inhibits the synthesis of:
A. ATP
B. GP 1
C. Prostaglandins
D. GP IIb and IIIa
L.O. 16.3
19. A platelet aggregation defect in which there is a deficiency of GP IIb/IIIa is known as:
A. vWD
B. Glanzmann’s thrombasthenia
C. Bernard-Soulier syndrome
D. TTP
L.O. 16.5
20. Platelets from patients with which two disorders will not aggregate with ristocetin?
A. Glanzmann’s syndrome and von Willebrand’s
B. Glanzmann’s syndrome and Chediak-Higashi
C. HUS and Bernard-Soulier syndrome
D. von Willebrand’s disease and Bernard-Soulier
L.O. 16.5
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